<?xml version="1.0" encoding="UTF-8"?><feed xmlns="http://www.w3.org/2005/Atom" xmlns:dc="http://purl.org/dc/elements/1.1/">
<title>Case Reports</title>
<link href="https://digitallibrary.bldedu.ac.in/xmlui/handle/123456789/4732" rel="alternate"/>
<subtitle/>
<id>https://digitallibrary.bldedu.ac.in/xmlui/handle/123456789/4732</id>
<updated>2026-08-15T07:29:17Z</updated>
<dc:date>2026-08-15T07:29:17Z</dc:date>
<entry>
<title>Management of traumatic tractional corectopia</title>
<link href="https://digitallibrary.bldedu.ac.in/xmlui/handle/123456789/6327" rel="alternate"/>
<author>
<name>Keerti Wali, Vaishnavi Patil, Vallabha K</name>
</author>
<id>https://digitallibrary.bldedu.ac.in/xmlui/handle/123456789/6327</id>
<updated>2026-08-05T12:32:36Z</updated>
<published>2026-02-01T00:00:00Z</published>
<summary type="text">Management of traumatic tractional corectopia
Keerti Wali, Vaishnavi Patil, Vallabha K
Corectopia is the displacement of the pupil. It can be&#13;
isolated congenital anamoly or associated with syndromes&#13;
like Ectopia lentis et pupillae or Axenfeld–Reiger anamoly.&#13;
[1] Aquired corectopia is often caused by trauma, surgery,&#13;
or anterior chamber inflammation.[2] It is caused by a&#13;
fibrous band exerting traction on the iris and may be&#13;
either stationary or progressive.[2] Treatment is tailored&#13;
considering the site of traction and associated comorbidities.&#13;
We present a case of traumatic tractional corectopia and its&#13;
management.
</summary>
<dc:date>2026-02-01T00:00:00Z</dc:date>
</entry>
<entry>
<title>Adult Adenoid Hypertrophy Mimicking a Nasopharyngeal Mass: A Diagnostic Challenge</title>
<link href="https://digitallibrary.bldedu.ac.in/xmlui/handle/123456789/6326" rel="alternate"/>
<author>
<name>Soumya Kori, Rupali Kumari , Savitri M. Nerune</name>
</author>
<id>https://digitallibrary.bldedu.ac.in/xmlui/handle/123456789/6326</id>
<updated>2026-08-05T12:24:47Z</updated>
<published>2026-03-01T00:00:00Z</published>
<summary type="text">Adult Adenoid Hypertrophy Mimicking a Nasopharyngeal Mass: A Diagnostic Challenge
Soumya Kori, Rupali Kumari , Savitri M. Nerune
The adenoids, also known as the nasopharyngeal tonsils, come from lymphoid tissue in the pharyngeal&#13;
mucosa. They are part of Waldeyer’s ring, which is a circular arrangement of lymphoid tissue. This ring&#13;
includes the pharyngeal (adenoid), palatine, lingual, and tubal tonsils. These tonsils are located at the&#13;
entrance of the aerodigestive tract.&#13;
They play an important role in mucosal immunity during early life by trapping inhaled pathogens and helping&#13;
with antigen presentation. This contributes to both humoral and cell-mediated immune responses.&#13;
Adenoid hypertrophy is predominantly a pediatric condition and rarely persists into adulthood due to&#13;
physiological involution after puberty. When present in adults, it may clinically and radiologically mimic&#13;
various nasopharyngeal pathologies, including malignancy.&#13;
We report a case of a 51-year-old woman presenting with long-standing left-sided nasal obstruction. She also&#13;
presented with snoring, mouth breathing and headache. Clinical examination revealed a deviated nasal
</summary>
<dc:date>2026-03-01T00:00:00Z</dc:date>
</entry>
<entry>
<title>Spontaneous occurrence and expulsion of a massive Steinstrasse.</title>
<link href="https://digitallibrary.bldedu.ac.in/xmlui/handle/123456789/5475" rel="alternate"/>
<author>
<name>Kundargi, Vinay</name>
</author>
<author>
<name>Patil, Santosh</name>
</author>
<author>
<name>Shukla, Vikas</name>
</author>
<author>
<name>Patil, Siddanagouda B</name>
</author>
<id>https://digitallibrary.bldedu.ac.in/xmlui/handle/123456789/5475</id>
<updated>2024-03-12T05:14:59Z</updated>
<published>2024-01-01T00:00:00Z</published>
<summary type="text">Spontaneous occurrence and expulsion of a massive Steinstrasse.
Kundargi, Vinay; Patil, Santosh; Shukla, Vikas; Patil, Siddanagouda B
</summary>
<dc:date>2024-01-01T00:00:00Z</dc:date>
</entry>
<entry>
<title>A Rare Case of Nicolau’s Syndrome (Embolia Cutis Medicamentosa) Following Intramuscular Diclofenac Sodium Injection in a Young Adult</title>
<link href="https://digitallibrary.bldedu.ac.in/xmlui/handle/123456789/5466" rel="alternate"/>
<author>
<name>M. S. Kotennavar, Aravind V. Patil</name>
</author>
<author>
<name>Pradeep Jaju, Narendra Ballal</name>
</author>
<id>https://digitallibrary.bldedu.ac.in/xmlui/handle/123456789/5466</id>
<updated>2024-03-08T07:19:24Z</updated>
<published>2024-01-01T00:00:00Z</published>
<summary type="text">A Rare Case of Nicolau’s Syndrome (Embolia Cutis Medicamentosa) Following Intramuscular Diclofenac Sodium Injection in a Young Adult
M. S. Kotennavar, Aravind V. Patil; Pradeep Jaju, Narendra Ballal
Nicolau syndrome (embolia cutis medicamentosa) is a condition where we see variable degrees of tissue necrosis including the skin and deeper tissues, due to an iatrogenic cause, intramuscular, subcutaneous, intra-articular injections, could be some of them. It occurs due to intravascular inoculation leading to crystal embolization causing thrombotic occlusion, vasospasm, and marked inflammation and surrounding necrosis. Here, we present a case of a 35-year-old gentleman who presented to us with bluish-purple discoloration of the skin followed by a necrotic patch and ulceration with multiple hyperpigmented spots over the back and lower limb post intramuscular diclofenac sodium injection. Routine investigation showed neutrophilia and thrombocytosis with increased creatinine. A diagnosis of Nicolau’s syndrome (embolia cutis medicamentosa) was made clinically, which was confirmed by histopathology following biopsy. Adequate surgical debridement and a combination therapy of analgesics, intravenous antibiotics, intravenous anticoagulants, and vasoactive therapy were administered. Split thickness skin grafting was done once the wound was healthy with rich granulation tissue, after 4 weeks of index surgery in this case. Patient recovered uneventfully.
</summary>
<dc:date>2024-01-01T00:00:00Z</dc:date>
</entry>
</feed>
