https://digitallibrary.bldedu.ac.in/xmlui/handle/123456789/4733 2026-05-20T10:58:10Z https://digitallibrary.bldedu.ac.in/xmlui/handle/123456789/5475 Spontaneous occurrence and expulsion of a massive Steinstrasse. Kundargi, Vinay; Patil, Santosh; Shukla, Vikas; Patil, Siddanagouda B 2024-01-01T00:00:00Z https://digitallibrary.bldedu.ac.in/xmlui/handle/123456789/5466 A Rare Case of Nicolau’s Syndrome (Embolia Cutis Medicamentosa) Following Intramuscular Diclofenac Sodium Injection in a Young Adult M. S. Kotennavar, Aravind V. Patil; Pradeep Jaju, Narendra Ballal Nicolau syndrome (embolia cutis medicamentosa) is a condition where we see variable degrees of tissue necrosis including the skin and deeper tissues, due to an iatrogenic cause, intramuscular, subcutaneous, intra-articular injections, could be some of them. It occurs due to intravascular inoculation leading to crystal embolization causing thrombotic occlusion, vasospasm, and marked inflammation and surrounding necrosis. Here, we present a case of a 35-year-old gentleman who presented to us with bluish-purple discoloration of the skin followed by a necrotic patch and ulceration with multiple hyperpigmented spots over the back and lower limb post intramuscular diclofenac sodium injection. Routine investigation showed neutrophilia and thrombocytosis with increased creatinine. A diagnosis of Nicolau’s syndrome (embolia cutis medicamentosa) was made clinically, which was confirmed by histopathology following biopsy. Adequate surgical debridement and a combination therapy of analgesics, intravenous antibiotics, intravenous anticoagulants, and vasoactive therapy were administered. Split thickness skin grafting was done once the wound was healthy with rich granulation tissue, after 4 weeks of index surgery in this case. Patient recovered uneventfully. 2024-01-01T00:00:00Z https://digitallibrary.bldedu.ac.in/xmlui/handle/123456789/5457 Port-wine stain associated with membranous aplasia cutis congenita and hair collar sign Inamadar, Arun 2024-01-01T00:00:00Z https://digitallibrary.bldedu.ac.in/xmlui/handle/123456789/4800 Coexistence of annular polycyclic, morpheaform and atrophic lesions in neonatal lupus erythematosus Kulkarni, Shruti; Adya, Keshavmurthy A; Janagond, Ajit B; Inamadar, Arun Neonatal lupus erythematosus (NLE) occurs due to transplacental transfer of autoantibodies in newborns of mothers with clinical or subclinical collagen vascular diseases. Anti-Ro/SSA antibodies are strongly associated with NLE. Anti-La/SSB and anti-U1 -RNP antibodies are less frequent. Cutaneous and cardiac manifestations are prominent of NLE. Nearly half of the cases show either cutaneous or cardiac features, and 10% show both. Skin lesions may be congenital or develop within 12–16 weeks postpartum 2022-01-01T00:00:00Z